CRISPR/Cas9 facilitates investigation of neural circuit disease using human iPSCs: mechanism of epilepsy caused by an SCN1A loss-of-function mutation
文献类型:期刊论文
作者 | Liu, J. ; Gao, C. ; Chen, W. ; Ma, W. ; Li, X. ; Shi, Y. ; Zhang, H. ; Zhang, L. ; Long, Y. ; Xu, H. ; Guo, X. ; Deng, S. ; Yan, X. ; Yu, D. ; Pan, G. ; Chen, Y. ; Lai, L. ; Liao, W. ; Li, Z. |
刊名 | TRANSLATIONAL PSYCHIATRY |
出版日期 | 2016 |
卷号 | 6 |
ISSN号 | 2158-3188 |
学科主题 | Psychiatry |
语种 | 英语 |
公开日期 | 2016-12-16 |
源URL | [http://ir.gibh.ac.cn/handle/344009/907] |
专题 | 广州生物医药与健康研究院_华南干细胞与再生医学研究所_华南干细胞与再生医学研究所_期刊论文 |
推荐引用方式 GB/T 7714 | Liu, J.,Gao, C.,Chen, W.,et al. CRISPR/Cas9 facilitates investigation of neural circuit disease using human iPSCs: mechanism of epilepsy caused by an SCN1A loss-of-function mutation[J]. TRANSLATIONAL PSYCHIATRY,2016,6. |
APA | Liu, J..,Gao, C..,Chen, W..,Ma, W..,Li, X..,...&Li, Z..(2016).CRISPR/Cas9 facilitates investigation of neural circuit disease using human iPSCs: mechanism of epilepsy caused by an SCN1A loss-of-function mutation.TRANSLATIONAL PSYCHIATRY,6. |
MLA | Liu, J.,et al."CRISPR/Cas9 facilitates investigation of neural circuit disease using human iPSCs: mechanism of epilepsy caused by an SCN1A loss-of-function mutation".TRANSLATIONAL PSYCHIATRY 6(2016). |
入库方式: OAI收割
来源:广州生物医药与健康研究院
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