The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction
文献类型：期刊论文
| 作者 | Chen Q ; Chai YC ; Mazumder S ; Jiang C ; Macklis R ; Chisolm GM ; Almasan A |
| 刊名 | Cell Death and Differentiation
 |
| 出版日期 | 2003 |
| 卷号 | 10期号:3页码:323-334 |
| 源URL | [http://ir.***.ac.cn/handle/000000/10393]  |
| 专题 | 膜生物学国家重点实验室 |
| 推荐引用方式 GB/T 7714 | Chen Q,Chai YC,Mazumder S,et al. The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction[J]. Cell Death and Differentiation,2003,10(3):323-334. |
| APA | Chen Q.,Chai YC.,Mazumder S.,Jiang C.,Macklis R.,...&Almasan A.(2003).The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction.Cell Death and Differentiation,10(3),323-334. |
| MLA | Chen Q,et al."The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction".Cell Death and Differentiation 10.3(2003):323-334. |
入库方式： OAI收割
来源：动物研究所
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