Effect of glaucocalyxin A on level of Th1/Th2 type cytokines in mice
文献类型：期刊论文
| 作者 | CHEN Zijun2; LI Yunsen1; ZHOU Jiyan2; WANG Zhengtao2 |
| 刊名 | China Journal of Chinese Materia Medica
 |
| 出版日期 | 2006 |
| 卷号 | 31期号:15页码:1257-1260 |
| 关键词 | Th1／Th2 glaucocalyxin A splenocytes Th1/Th2 cytokine |
| ISSN号 | 1001-5302 |
| 其他题名 | 蓝萼甲素对小鼠脾细胞Th1／Th2型5种细胞因子水平的影响 |
| 文献子类 | Article |
| 英文摘要 | Objective： Toresearch the effect of glaucocalyxinA（GLA） on the level of Th1/Th2 type cytokines in mice. Method： By using flow cytometer with CBA software to detect Th1/Th2 type cytokines. Result： GLA had insighificant inhibitory effects on Th1 and Th2 cytokines （IL-2, IFN-gamma,TNF-alpha,IL-4 and IL-5） induced by ConA, in which more potential on cytokines from Th1 than three of from Th2 were displayed. However, GLA could produce inhibition on IL-2, IFN-gamma and TNF-alpha and acceleration on IL-4 and IL-5. Conclusion： Immunosuppressive effect of GLA is related to the influence the level of Th1/Th2 type cytokines. |
| 资助项目 | 国家科技部863计划新药博士基金项目[00000000] |
| WOS研究方向 | General & Internal Medicine (provided by Clarivate Analytics) |
| 语种 | 中文 |
| CSCD记录号 | CSCD:2462778 |
| 源URL | [http://119.78.100.183/handle/2S10ELR8/268175]  |
| 专题 | 信息中心 药理学第二研究室 药理学第一研究室 |
| 作者单位 | 1.Shanghai Institute of Materia Medica, Chinese Academy of Scion, Shanghai 201203, China. 2.Institute of Chinese Meteria Medica, Shanghai University of Traditional Chinese Medicine, Shanghai 201203, China.; |
| 推荐引用方式 GB/T 7714 | CHEN Zijun,LI Yunsen,ZHOU Jiyan,et al. Effect of glaucocalyxin A on level of Th1/Th2 type cytokines in mice[J]. China Journal of Chinese Materia Medica,2006,31(15):1257-1260. |
| APA | CHEN Zijun,LI Yunsen,ZHOU Jiyan,&WANG Zhengtao.(2006).Effect of glaucocalyxin A on level of Th1/Th2 type cytokines in mice.China Journal of Chinese Materia Medica,31(15),1257-1260. |
| MLA | CHEN Zijun,et al."Effect of glaucocalyxin A on level of Th1/Th2 type cytokines in mice".China Journal of Chinese Materia Medica 31.15(2006):1257-1260. |
入库方式： OAI收割
来源：上海药物研究所
浏览0
下载0
收藏0
其他版本
除非特别说明，本系统中所有内容都受版权保护，并保留所有权利。