中国科学院机构知识库网格
Chinese Academy of Sciences Institutional Repositories Grid
Reactive oxygen species contribute to oridonin-induced apoptosis and autophagy in human cervical carcinoma HeLa cells

文献类型:期刊论文

作者Zhang Yahong1; Wu Yingliang1; Tashiro Shinichi3; Onodera Satoshi3; Ikejima Takashi1
刊名ACTA PHARMACOLOGICA SINICA
出版日期2011
卷号32期号:10页码:1266-1275
关键词MITOCHONDRIAL PERMEABILITY TRANSITION OXIDATIVE STRESS MEDIATED AUTOPHAGY CANCER DEATH ACTIVATION MECHANISM EXPRESSION GENERATION INDUCTION oridonin reactive oxygen species apoptosis autophagy Bax Bcl-2 caspase 3 caspase 8 HeLa cell
ISSN号1671-4083
其他题名Reactive oxygen species contribute to oridonin-induced apoptosis and autophagy in human cervical carcinoma HeLa cells
英文摘要Aim: To investigate the role of reactive oxygen species (ROS) in oridonin-induced apoptosis and autophagy in HeLa cells.
资助项目[China Postdoctoral Science Foundation] ; [He-nan University]
语种英语
CSCD记录号CSCD:4335577
源URL[http://ir.imr.ac.cn/handle/321006/143425]  
专题金属研究所_中国科学院金属研究所
作者单位1.中国科学院金属研究所
2.河南大学
3.Showa Pharmaceut University, Dept Clin & Biomed Sci, Tokyo 1948543, Japan
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GB/T 7714
Zhang Yahong,Wu Yingliang,Tashiro Shinichi,et al. Reactive oxygen species contribute to oridonin-induced apoptosis and autophagy in human cervical carcinoma HeLa cells[J]. ACTA PHARMACOLOGICA SINICA,2011,32(10):1266-1275.
APA Zhang Yahong,Wu Yingliang,Tashiro Shinichi,Onodera Satoshi,&Ikejima Takashi.(2011).Reactive oxygen species contribute to oridonin-induced apoptosis and autophagy in human cervical carcinoma HeLa cells.ACTA PHARMACOLOGICA SINICA,32(10),1266-1275.
MLA Zhang Yahong,et al."Reactive oxygen species contribute to oridonin-induced apoptosis and autophagy in human cervical carcinoma HeLa cells".ACTA PHARMACOLOGICA SINICA 32.10(2011):1266-1275.

入库方式: OAI收割

来源:金属研究所

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