还原性无机硫  活性铁  深层柱状沉积物  硫化度  矿化度  Reduced inorganic sulfur  Reactive iron  Deep core sediments  Degree of pyritization  Degree of sulfidity  The distribution characteristics and coupling mechanism of reduced inorganic sulfur (RIS)and reactive iron in marine core sediments are closely related to the evolution of environment quality.In this study,an improved cold diffusion method and hydrochloric acid extraction method were applied to the acid-volatile sulfur (AVS),pyrite sulfur (CRS),elemental sulfur (ES),and reactive iron [Fe (Ⅱ)and Fe (III)] analyses.The distribution characteristics and coupling mechanism of S and Fe were investigated based on a core sediment 4 meters deep collected in the northern sea area of the mouth of Jiahe River,Yantai.The results showed that the RIS in sediments was dominated by CRS,followed by AVS and ES.The content of AVS presented a narrow range with depth,whereas the CRS and ES were higher at the top and bottom layers than the middle layer  reactive iron was dominated by Fe (II),which increased with depth,while Fe (Ⅲ)gradually decreased with depth.Most of the Fe (III)was reduced to the disssolved Fe(II),which was combined with the soluble H_2S of the sulfate-reducing bacteria to produce CRS and ES in the deep layer,resulting in their accumulation at the bottom of core sediments.Furthermore,this study showed that reactive iron was not a limiting factor for the accumulation of RIS with lower degree of pyritization and degree of sulfidity.  

TiO2 nanoparticles (TiO2 NPs)  arsenic [As(III)]  human-hamster hybrid (A(L)) cells  synergistic genotoxicity  physicochemical interactions  mitochondria  

active zone stability  Drosophila  neuromuscular junction  dephosphorylation  Liprin-α  Syd-1  PP2A  GSK-3?  living scaffolds  neural tissue engineering  cell transplant  biomaterials  regeneration  neurotrauma  neurodegeneration  axon pathfinding  cell migration  injury  plasticity  neurodegenerative disease  brain  therapy  neuron  microglia  neural progenitor  tissue engineering  neuroregeneration  repair  central nervous system  biomaterial  regenerative medicine  nanotechnology  spinal cord injury  axonal regeneration  exosome  extracellular vesicle  microRNA  microvesicle  nerve gap  neurite outgrowth  peripheral nerve injury  Schwann cell  stem cell  hemodynamic phases  cerebral subarachnoid hemorrhage  metabolic crises  nerve regeneration  hypoxic-ischemic brain damage  ginsenoside Rg1  neural stem cells  cell transplantation  cell differentiation  cognition  nerve reconstruction  neural regeneration  nerve regeneration  brain injury  neuroimaging  functional magnetic resonance imaging  regional homogeneity  apoplexy  subacute  ischemia  participants  healthy  volunteers  brain activity  NSFC grants  neural regeneration  nerve regeneration  brain injury  neuroprotection  cerebral ischemia/reperfusion injury  lateral intracerebroventricular injection  Apelin-13  nerve apoptosis  Bcl-2  caspase-3  NSFC grants  neural regeneration  nerve regeneration  fractalkine  CX3 chemokine receptor 1  neuronal maturation  dendrites  doublecortin  synaptic maturation  newborn neurons  neural regeneration  nerve regeneration  neurodegenerative diseases  Alzheimer′s disease  transgenic animal models  mice  epimedium herb  milkvetch root  kudzuvine root  divalent metal transporter 1  ferroportin 1  neural regeneration  nerve regeneration  microRNA-124  neurogenesis  neuronal survival  Huntington′s disease  SRY-related HMG box transcription factor 9  brain-derived neurotrophic factor  peroxisome proliferator-activated receptor gamma coactivator 1-alpha  mutant huntingtin  nerve regeneration  Ras/Raf/Erk1/2 signaling pathway  spinal cord injury  apoptosis  repair  regulation  inhibition  neural regeneration  nerve regeneration  neurogenic bladder  bibliometric analysis  Web of Science database  visualization analysis  CiteSpace III  citation analysis  neural regeneration  nerve regeneration  sciatic nerve injury  autologous nerve grafting  epineurial suturing  three-dimensional finite element models  load  stress  displacement  neural regeneration  nerve regeneration  brain injury  facial palsy  Bell′s palsy  comparison  methodological quality  therapy  fixed effect model  acupuncture  incomplete recovery  randomized controlled trials  electroacupuncture  NSFC grants  neural regeneration  nerve regeneration  peripheral nerve injury  thalidomide  ankylosing spondylitis  adverse reactions  peripheral neuropathy  treatment  dose  treatment time  age  sex  neural regeneration  Neurology. Diseases of the nervous system  RC346-429  prospective study  

一种手霉素生物合成基因簇  编码3?氨基?4?羟基?苯甲酸(3  编码醛裂合酶的chiA3  编码3?羟丁酰辅酶A脱氢酶的chiB1  编码芳香胺N乙酰转移酶的chiB2  编码3?氧乙酰基载体蛋白合成酶III的chiB3  编码3?氧乙酰基载体蛋白合成酶III的chiB4  编码酰基载体蛋白的chiB5  编码硫酯酶的chiB6  编码3?氧乙酰基载体蛋白还原酶的chiB7  编码酰基脱水酶的chiB8  编码酰基脱水酶的chiB9  编码I/II型酮合成酶相关的酰基载体蛋白的chiC1  编码3?氨基?4?羟基?苯甲酸(3  编码3?氧乙酰基载体蛋白合成酶II的chiC3  编码酮合成酶的chiC4  编码甲基丙二酰辅酶A变位酶的chiC5  编码硫酯酶的chiC6  编码酰胺合成酶的chiD1  编码5?氨乙酰丙酸合成酶的chiD2  编码5?氨乙酰丙酸辅酶A连接酶的chiD3  编码氧化酶的chiE1  编码黄素还原酶的chiE2  编码黄素依赖的单加氧酶的chiE3  编码黄素依赖的氧化还原酶的chiE4  共25个基因  2)手霉素A和中尼霉素的生物合成的调节基因：编码LuxR家族转录调节子的chiR1  编码TetR家族转录调节子的chiR2  编码调节蛋白的chiR3  共3个基因  3)手霉素A和中尼霉素的生物合成的转运基因：编码抗药性转运蛋白的chiM1  编码分泌蛋白酶的chiM2  共2个基因。  其特征在于：所述手霉素生物合成基因簇碱基序列如SEQ?ID?NO.1所示  4?AHBA)羧基腺苷酰蛋白的chiA2  4?AHBA)载体蛋白的chiC2  其中手霉素生物合成基因簇为手霉素A和中尼霉素的生物合成基因簇  1)手霉素A和中尼霉素的生物合成的结构基因：编码3?氨基?4?羟基?苯甲酸(3  4?AHBA)合成酶的chiA1  

a fluorine functionalized 1  10-phenanthroline ligand  3-ethyl-2-(2- fluorophenyl) imidazo[4  5-f]1  10-phenanthroline(EFPIP) and the corresponding red-emitting Eu(III) complex Eu(DBM)3(EFPIP) (DBM = dibenzoylmethanate) was synthesized and the photophysical properties and electroluminescent(EL) performances were researched. When Eu(DBM) 3(EFPIP) was used as emitting material in organic devices  the maximum efficiency and luminance of red emission achieved from the EL device with the configuration of ITO/m-MTDATA (30 nm)/NPB (20 nm)/x wt% Eu(DBM) 3(EFPIP) doped in CBP (30 nm)/Bphen (20 nm)/Alq3 (20 nm)/LiF (0.8 nm)/Al were 3.6 cd/A and 563 cd/m2  respectively. Compared with the previously reported devices based on methyl or chlorine substitute Eu(III) complexes  the EL performances of the device using Eu(DBM)3(EFPIP) as an emitter was significantly enhanced due to the introduction of fluorine. (2012) Trans Tech Publications  Switzerland.