中国科学院机构知识库网格
Chinese Academy of Sciences Institutional Repositories Grid
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CAS IR Grid
机构
上海神经科学研究所 [2]
高能物理研究所 [1]
上海生物化学与细胞生... [1]
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OAI收割 [4]
内容类型
期刊论文 [4]
发表日期
2014 [1]
2012 [1]
2003 [1]
2002 [1]
学科主题
Neuroscien... [4]
Physiology [1]
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学科主题:Neurosciences & Neurology
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Reductions of the Components of the Calreticulin/Calnexin Quality-Control System by Proteasome Inhibitors and Their Relevance in a Rodent Model of Parkinson's Disease
期刊论文
OAI收割
JOURNAL OF NEUROSCIENCE RESEARCH, 2014, 卷号: 92, 期号: 10, 页码: 1319-1329
作者:
Kuang, XL
;
Liu, F
;
Chen, HF
;
Li, YP
;
Liu, YM
收藏
  |  
浏览/下载:25/0
  |  
提交时间:2015/07/22
ubiquitin-proteasome system (UPS)
unfolded protein response (UPR)
chaperone
calreticulin/calnexin cycle
ER quality control
endoplasmic reticulum stress
endoplasmic reticulum-associated degradation (ERAD)
Parkinson's disease
Acupuncture regulates the glucose metabolism in cerebral functional regions in chronic stage ischemic stroke patients-a PET-CT cerebral functional imaging study
期刊论文
OAI收割
BMC NEUROSCIENCE, 2012, 卷号: 13, 页码: 75
作者:
Huang, Y
;
Tang, CZ
;
Wang, SX
;
Lu, YJ
;
Shen, W
收藏
  |  
浏览/下载:23/0
  |  
提交时间:2016/04/08
Waiguan (TE5)
Sham point
Ischemic stroke
Needling/sham needling
PET-CT cerebral functional imaging
Cerebral activating/deactivating effect
Na+ channel inactivation: a comparative study between pancreatic islet beta-cells and adrenal chromaffin cells in rat
期刊论文
OAI收割
JOURNAL OF PHYSIOLOGY-LONDON, 2003, 卷号: 548, 期号: 1, 页码: 191-202
Lou, XL
;
Yu, X
;
Chen, XK
;
Duan, KL
;
He, LM
;
Qu, AL
;
Xu, T
;
Zhou, Z
收藏
  |  
浏览/下载:13/0
  |  
提交时间:2012/07/23
B-CELLS
INSULIN-SECRETION
ACTION-POTENTIALS
PATCH-CLAMP
QUANTAL SECRETION
POTASSIUM CHANNEL
IONIC CURRENTS
CA2+ CURRENT
CALCIUM
SODIUM
Evaluation of beta-amyloid peptide 25-35 on calcium homeostasis in cultured rat dorsal root ganglion neurons
期刊论文
OAI收割
BRAIN RESEARCH, 2002, 卷号: 939, 期号: 40910, 页码: 65-75
He, LM
;
Chen, LY
;
Lou, XL
;
Qu, AL
;
Zhou, Z
;
Xu, T
收藏
  |  
浏览/下载:32/0
  |  
提交时间:2012/07/23
beta-amyloid peptide
calcium channel
calcium store
ALZHEIMERS-DISEASE
INTRACELLULAR CA2+
PRECURSOR PROTEIN
CHANNEL FORMATION
OXIDATIVE STRESS
CORTICAL-NEURONS
CELLS
NEURODEGENERATION
MECHANISM
FORMS